Preprint established that higher concentrations of Galectin-9 led to more infection in terms of SARS-CoV-2 infected cells. The concept is that the Galectin-9 facilitates viral entry at a significantly higher rate than the control. In vitro studies also factored in and measured the expression of TMPRSS and ACE2 for a proper comparison. Reference to Sigamani et al study.

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Human Galectin-9 Potently Enhances SARS-CoV-2 Replication and Inflammation In Airway Epithelial Cells

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Human Galectin-9 Potently Enhances SARS-CoV-2 Replication and Inflammation In Airway Epithelial Cells

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